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12/7/08
Patient Airway – No airway, no patient
Upper Airway – Major function, warmer, filter and humidly air.
Turbinates – three bony shelves in nasal passage – increases surface area
Sinuses – Cavities formed by the cranial bones
- Fractures of these bones may cause cerebrospinal fluid to leak from the nose or the ears.
- Significant bleeding from sinus fractures
- Prevent contaminants from entering the respiratory tract
LeFort Fractures – facial fractures
Hyoid bone, beneath mandible, anchors tongue, only non articulating bone in the body, attached with tendons
Palate – Hard and Soft
Adenoids and tonsils : lymphatic tissue that filter bacteria, severe swelling can cause airway obstruction
Vallecula – between base of tongue and epiglottis –It is an important landmark in intubation;
Larynx – divides upper and lower airways
Lower airway
- Externally from 4th cervical vertebrae to xyphoid process
- Trachia,(enters lung at hilum) Bronchi, smaller bronchi, bronchioles, alveoli (functional site for respiration (i.e. gas exchange)
- Trachia bifurcates at the Carina
- R has 3 lobes, L has 2 lobes
- Visceral pleura and parietal pleura
- Ventilation vs Respiration
- Shunting (blood bypasses the alveoli and does not exchange gasses)
- Alveoli lined with surfactant
- Atelectasis
- Avg adult 6L of air volume
- Tidal volume (Vt), 5-7Ml/kg adult, 6-8mL/kg (500 mL)
- Alveolar volume = tidal volume minus dead space(Vd)
- Minute volume (Vm)– amount of air that passes over a minute (Vt – Vd x Resp Rate)
- Functional reserve capacity – amount you can force out in single exhalation – (about 1200mL)
- Ventilation- Inhalation 1/3rd Exhalation 2/3
Regulation of ventilation
- Primary reglated by PH levels in CSF
- Phrenic nerve- innervates the diaphragm
- Intercostals nerves – innervates the intercostals muscles
- Hering-Bruer reflex –stretch receptors tells the brain that the chest wall is fully expanded – ending inspiration
- Apneustic center – increase respiratory rate
- Pneumotaxic center – counters by inhibiting inspiration
- Chemoreceptors – monitor CO2, O2 and PH levels
- Respiratory drive – primary drive – driven by CO2
- Hypoxic drive – secondary drive driven by O2 (seen in COPD patients)
- Inhalation, muscles contract, pressure decreases – move air into lungs via negative pressure
- Exhalation, muscles relax – pressure increases, air forced out of lungs (passive)
Respiration
- Diffusion
- SaO2 measures saturation of hemoglobin, can be O2 or Co2 – use a pulse oximeter
- Hypercarbia
- Hypocarbia
Abnormal CO2 Concentrations
- As metabolic rate goes up more CO2 is produced
- Hypoventilation – co2 production exceeds body’s ability to eliminate it (hypercarbia)
- Hyperventilation – co2 elimination exceeds production (hypocarbia)
- Minute volume – decreased minute volume causes a decrease in co2 elimination
ET Tube
- Sizes 5.5 up have pilot cuff –
- 5.5 and down are cuffless – for peds
- Age + 16 / 4 = tube size (pediatrics)
- Keep spo2 above 90, hyperventilate before attempts
ETT Verification
- Seeing pass through cords
- Moisture in tube
- Etco2
- Auscultate lungs and abdomen
- Esophageal detector
Tape and secure. Put an oral airway in to hold tongue and prevent biting, record placement level