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Orotracheal Intubation – Medical Procedures – Medstudents
The height of the table where the patient is lied, should be adjusted so that the patient’s face is at the level of the xiphoid cartilage of the standing person who is performing the procedure. Elevating the patient’s head about 10 cm with pads under the occiput and extension of the head at the atlanto-occipital joint (sniffing position) serve to align the oral, pharyngeal, and laryngeal axis, so that the passage from the lips to the glottic opening is almost a straight line. This position permits better visualization of the glottis and vocal cords and allows easier passage of the endotracheal tube. For children under 1 month of age, the head should be in a neutral position. See Figure 1.
Figure 1:Letter A shows the wrong and letter B shows the correct position of patient’s head.
Some exam review for Respiratory emergencies:
1. A person who experiences sharp chest pain followed by increasing dyspnea after he or she coughs MOST likely has:
Choose one answer.
A. pleurisy.
B. acute pneumonia.
C. a pleural effusion.
D. a pneumothorax.
2. An otherwise healthy adult whose normal hemoglobin level is 12 to 14 g/dL typically will begin to exhibit cyanosis when: Choose one answer. A. hemoglobin levels fall below 12 g/dL. B. about 5 g/dL of hemoglobin is desaturated. C. his or her oxygen saturation falls below 50%. D. 10% of his or her hemoglobin is desaturated.
2. An otherwise healthy adult whose normal hemoglobin level is 12 to 14 g/dL typically will begin to exhibit cyanosis when:
A. hemoglobin levels fall below 12 g/dL.
B. about 5 g/dL of hemoglobin is desaturated.
C. his or her oxygen saturation falls below 50%.
D. 10% of his or her hemoglobin is desaturated.
3. Intrapulmonary shunting occurs when:
A. hyperinflated alveoli retain high levels of carbon dioxide.
B. resistance to airflow increases due to bronchoconstriction.
C. nonfunctional alveoli inhibit pulmonary gas exchange.
D. the volume of anatomic dead space suddenly increases.
4. A patient with status asthmaticus commonly presents with: Choose one answer. A. compensatory respiratory alkalosis and stridor. B. physical exhaustion and inaudible breath sounds. C. audible expiratory wheezing and severe cyanosis. D. accessory muscle use and inspiratory wheezing.
4. A patient with status asthmaticus commonly presents with:
A. compensatory respiratory alkalosis and stridor.
B. physical exhaustion and inaudible breath sounds.
C. audible expiratory wheezing and severe cyanosis.
D. accessory muscle use and inspiratory wheezing.
5. __________ breath sounds are the MOST commonly heard breath sounds, and have a much more obvious inspiratory component.
A. Vesicular
B. Tracheal
C. Bronchovesicular
D. Bronchial
6. A 29-year-old woman is experiencing a severe asthma attack. Her husband reports that she was admitted to an intensive care unit about 6 months ago, and had a breathing tube in place. Prior to your arrival, the patient took 3 puffs of her rescue inhaler without effect. She is anxious and restless, tachypneic, and has audible wheezing. You should: Choose one answer. A. attempt to slow her breathing with respiratory coaching, administer a nebulized bronchodilator, and transport. B. start an IV of normal saline, administer methylprednisolone via IV push, and transport as soon as possible. C. apply a CPAP unit, transport immediately, and attempt to establish vascular access en route to the hospital. D. begin assisting her ventilations with a bag-mask device and 100% oxygen and prepare to intubate her trachea.
6.
A 29-year-old woman is experiencing a severe asthma attack. Her husband reports that she was admitted to an intensive care unit about 6 months ago, and had a breathing tube in place. Prior to your arrival, the patient took 3 puffs of her rescue inhaler without effect. She is anxious and restless, tachypneic, and has audible wheezing. You should:
A. attempt to slow her breathing with respiratory coaching, administer a nebulized bronchodilator, and transport.
B. start an IV of normal saline, administer methylprednisolone via IV push, and transport as soon as possible.
C. apply a CPAP unit, transport immediately, and attempt to establish vascular access en route to the hospital.
D. begin assisting her ventilations with a bag-mask device and 100% oxygen and prepare to intubate her trachea.
7. The presence of diffuse rhonchi in the lungs indicates:
A. thick secretions in the large airways.
B. isolated consolidation of secretions.
C. right-sided congestive heart failure.
D. air being forced through narrowed airways.
8. Uncontrollable coughing and hemoptysis in a cigarette smoker are clinical findings MOST consistent with: Choose one answer. A. acute bronchitis. B. lung cancer. C. emphysema. D. pleural effusion.
8. Uncontrollable coughing and hemoptysis in a cigarette smoker are clinical findings MOST consistent with:
A. acute bronchitis.
B. lung cancer.
C. emphysema.
D. pleural effusion.
Dr Hernandez 3/1/09 Pulmonary Embolism Pulmonary artery blockage Risk factors 5 Fs – female, fat, forty, fertile, fair Usually present in the acute with normal lung sounds After that area of lung becomes ischemic it may start wheezing and then after a while – alveolar collapse and absent lung sounds Saddle embolism – immediately fatal Pain with walking , diameter of one leg bigger than other Usually will not travel far enough to cause CVA as gets trapped in lungs Contributing factors Venous injury, venostasis, increased coagulability (triad) Pregnancy (blood loss, tearing of placenta, hypercoagulability) Disease (cancer, tumors, Multiple trauma, hemostasis Immobility after surgery, other prolonged immobility PE Assessment Cough Hemoptysis (rare) Pain Anxiety Syncope Hypotension Pleural rub Tachycardia Tachypnea Fever (rare) JVD Localized crackles, wheezing PE Management O2 EKG Monitoring Pulse Oximeter IV NS or LR Rapid Transport URI Nose throat sinuses larynx Cold pharyngitis tonsillitis sinusitis laryngitis croup Rarely life threatening hand washing – key beware if immunocompromised, transplant patent, infants Some other pediatric lower respiratory infections Bronchiolitis also see AAP Policy RSV – Respiratory Syncytial Virus Spontaneous Pneumothorax dyspnea , chest pain, pallor, diaphoresis, tachypnea Tension Pneumothorax Absent one side , greatly diminished on other JVD, muffled heart tones, tracheal deviation Pharmacological Management Oxygen Albuterol – asthma, copd, anaphylaxis Epinephrine ipratropium – Anticholinergic – caution with peanut allergies Isoetharine Alupent - metaproterenol Racemic epinephrine – upper airway – nebulized – croup Terbutaline Magnesium sulfate Corticosteroids ARDS – common causes Always a precipitating event Sepsis Bronchial aspiration of gastric contents Multiple trauma Inhalation – fires 12-48 hours for S&S Young people with no medical Hx Can occur in peds Cystic Fibrosis Thick secretions Can lead to many other respiratory problems More discussion about EKG machines and ECG monitoring will be discussed in future lectures.
Dr Hernandez
3/1/09
Pulmonary Embolism
Contributing factors
PE Assessment
PE Management
URI
Some other pediatric lower respiratory infections
Spontaneous Pneumothorax
Tension Pneumothorax
Pharmacological Management
ARDS – common causes
Cystic Fibrosis
More discussion about EKG machines and ECG monitoring will be discussed in future lectures.
Dr Hernandez 2/25/09 Pathophysiology of Ventilation Problems Upper Airway Trauma Epiglottis Croup FBAO Tonsillitis Lower Airway Trauma Blunt Penetrating Obstructive Lung Disease – (COPD; emphysema and chronic bronchitis, asthma – smooth muscle spasm) Restrictive lung disease – loss of lung compliance causing incomplete lung expansion and increased lung stiffness. Tension Pneumothorax Pneumo/hemo thorax Acute Respiratory Distress Syndrome (ARDS)(bruised lungs, ecchymotic, swelling etc) Airway edema, anaphylaxis, smoke inhalation, APE Empyema – puss purulent material secondary to pneumonia Plural Inflammation – will cause pleuritic chest pain CF, MS Neurologic problems Depressants CVA Head problems Phrenic or spinal nerve dysfunction Airway burns – soot around the mouth Asbestosis Blebs PE –(off plane, long period of immobility, DVT, recent surgery) Interstitial Space Pathophysiology Pulmonary Edema ARDS Near Drowning – freshwater vs. saltwater – Freshwater can be more dangerous than saltwater in secondary drowning. When fresh water enters the lungs it is pulled into the pulmonary circulation via the alveoli because of the low capillary hydrostatic pressure and high colloid osmotic pressure. Consequently, the plasma is diluted and the hypotonic environment causes red blood cells to burst (hemolysis). The resulting elevation of plasma K+ level and depression of Na+ level, due to the hemolysis, alter the electrical activity of the heart. Ventricular fibrilation often occurs as a result of these electrolyte changes. Additionally, if drowning occurs in very cold water ( <10o C), the uptake of cold water into the vascular system can stop the heart. In open heart surgery, the technique of pouring cold saline solution over the heart is used to prevent heart action. If the victim is resuscitated death can occur hours later due to renal failure. During hemolysis, hemoglobin is also released into the plasma which can accumulate in the kidneys leading to acute renal failure. In contrast, salt-water drowning does not lead to uptake of inspired water into the vascular system because it is isotonic to blood. Therefore, no red cell hemolysis occurs and the cause of death is asphyxia. Max Lung Capacity – 6L Dead space – 150ml Tidal volume 5-6 ml/kg – 6-8ml/kg for peds Minute volume Alveolar Volume Inspiratory reserve volume, max amount that can be inhaled Forced Expiratory Volume (FEV) Residual Volume – amt remaining in lung after exhalation Acid Base Balance Normal ph 7.35-7.45 – out of this range normal metabolic process cannot occur properly, acidosis more harmful and more acute than alkalosis (3 mechanisms of H+ removal – kidneys hold on to bicarb) Decreased ventilation = Retention of co2 = acidosis Hyperventilate = blow off co2 = alkalosis = nerve ending malfunction, tingling, carpopedal spasm The Bicarbonate buffering system is the most important buffer solution for maintaining a relatively constant pH in the plasma. It functions via blood buffering with bicarbonate. The enzyme carbonic anhydrase maintains the equilibrium between bicarbonate and H2CO3. This is, in turn, converted into carbon dioxide and water. Most common cause of hemoptysis, chronic bronchitis, blebs, lung CA, TB – The origin of blood can be known by observing its color. Bright red, foamy blood comes from the respiratory tract while dark red, coffee-colored blood comes from the gastrointestinal tract. COPD - Blue Bloaters vs Pink Puffers (The effort made by patients suffering from emphysema during exhalation, causes a pink color in their faces, hence the term commonly used to refer to them, “pink puffers”.) (Patients with advanced COPD that have primarily chronic bronchitis rather than emphysema were commonly referred to as “blue bloaters” because of the bluish color of the skin and lips (cyanosis) seen in them.) Emphysema – destruction of alveolar walls – Chronic bronchitis – too much mucus production The Pores of Kohn are pores between adjacent alveoli, or interalveolar connections. They function as a means of collateral ventilation; that is, if the lung is partially deflated, ventilation can occur to some extent through these pores. The pores also allow the passage of other materials such as fluid and bacteria. Pursed lips breathing – patient is building pressure to alveoli to help breathe. Self PEEPing Asthma Reactive airway disease More common in peds and young-middle adults Triggers – Extrinsic in Peds, Intrinsic in adults Reversible airflow obstruction due to: Bronchial smooth muscle constriction Overproduction of mucus Breath Stacking – can’t exhale complete volume – leads to hyperinflation of alveoli Breath trapping – physical finding, chest gets bigger Bronchiole is obstructed on expiration – mucus plugs Hypertrophy of smooth muscle in asthmatics vs COPD Assessment Inspiratory wheezing – larger airways Expiratory wheezing – Silent chest may mean severe obstruction – flow too low to generate sounds – BAD! As lungs are hyperinflated – cause symptoms of cardiac tamponade – narrow pulse pressure – pulsus paradoxus>20 As they get better they may start wheezing and bringing up mucus – as bronchodilation is occurring Status Asthmaticus – severe and prolonged – taking his meds all day, etc. True Emergency – Up Triage – imminent threat of respiratory failure Role of Magnesium Sulfate in Asthma, Maged A. Zaki, MD in Medscape Magnesium sulfate has been shown to inhibit smooth muscle contraction, decrease histamine release from mast cells, and inhibit acetylcholine release. Studies both in children and adults have shown variable improvement in patients with severe airflow limitation who are unresponsive to standard treatment with beta agonist, anticholinergic, and corticosteroid medications. Ciarallo has shown in 2 studies of children that the optimum dose is 40 mg/kg given as an intravenous bolus with a maximum dose of 2 g. In 1 study, improvement in forced expiratory volume in 1 second (FEV1) was seen within 20 minutes and continued for up to 110 minutes. Up to a 20% improvement in FEV1 may be seen. In adults, a study by Silverman has shown that patients with an initial FEV1 lower than 25% of what was predicted benefited the most from magnesium sulfate therapy. Asthma is classified according to the frequency of symptoms, FEV1 and peak expiratory flow rate.[4] Classification of asthma severity[4] Severity Symptom frequency Nighttime symptoms Peak expiratory flow rate or FEV1 of predicted Variability of peak expiratory flow rate or FEV1 Intermittent < once a week ≤ twice per month ≥ 80% predicted < 20% Mild persistent > once per week but < once per day > twice per month ≥ 80% predicted 20–30% Moderate persistent Daily > once per week 60–80% predicted > 30% Severe persistent Daily Frequent < 60% predicted > 30% Bacterial Pneumonia Fever , chills, pleuritic chest pain, dyspnea Empyema Decreased sounds 02 and transport Some more study resources from Wikipedia Respiratory sounds: Stridor ·Wheeze ·Rales ·Rhonchi ·Hamman’s sign Dyspnea ·Hyperventilation/Hypoventilation ·Hyperpnea/Tachypnea/Bradypnea ·Orthopnoea/Platypnea Biot’s respiration ·Cheyne-Stokes respiration ·Kussmaul breathing Hiccup ·Mouth breathingAsphyxia ·Cough ·Pleurisy ·Sputum Respiratory arrest ·Hypercapnia/Hypocapnia
2/25/09
Pathophysiology of Ventilation Problems
Upper Airway
Lower Airway
Interstitial Space Pathophysiology
Additionally, if drowning occurs in very cold water ( <10o C), the uptake of cold water into the vascular system can stop the heart. In open heart surgery, the technique of pouring cold saline solution over the heart is used to prevent heart action. If the victim is resuscitated death can occur hours later due to renal failure. During hemolysis, hemoglobin is also released into the plasma which can accumulate in the kidneys leading to acute renal failure. In contrast, salt-water drowning does not lead to uptake of inspired water into the vascular system because it is isotonic to blood. Therefore, no red cell hemolysis occurs and the cause of death is asphyxia.
Acid Base Balance
Most common cause of hemoptysis, chronic bronchitis, blebs, lung CA, TB – The origin of blood can be known by observing its color. Bright red, foamy blood comes from the respiratory tract while dark red, coffee-colored blood comes from the gastrointestinal tract.
COPD - Blue Bloaters vs Pink Puffers (The effort made by patients suffering from emphysema during exhalation, causes a pink color in their faces, hence the term commonly used to refer to them, “pink puffers”.) (Patients with advanced COPD that have primarily chronic bronchitis rather than emphysema were commonly referred to as “blue bloaters” because of the bluish color of the skin and lips (cyanosis) seen in them.)
The Pores of Kohn are pores between adjacent alveoli, or interalveolar connections. They function as a means of collateral ventilation; that is, if the lung is partially deflated, ventilation can occur to some extent through these pores. The pores also allow the passage of other materials such as fluid and bacteria.
Pursed lips breathing – patient is building pressure to alveoli to help breathe. Self PEEPing
Asthma
Assessment
Status Asthmaticus – severe and prolonged – taking his meds all day, etc. True Emergency – Up Triage – imminent threat of respiratory failure
Role of Magnesium Sulfate in Asthma, Maged A. Zaki, MD in Medscape
Magnesium sulfate has been shown to inhibit smooth muscle contraction, decrease histamine release from mast cells, and inhibit acetylcholine release. Studies both in children and adults have shown variable improvement in patients with severe airflow limitation who are unresponsive to standard treatment with beta agonist, anticholinergic, and corticosteroid medications. Ciarallo has shown in 2 studies of children that the optimum dose is 40 mg/kg given as an intravenous bolus with a maximum dose of 2 g. In 1 study, improvement in forced expiratory volume in 1 second (FEV1) was seen within 20 minutes and continued for up to 110 minutes. Up to a 20% improvement in FEV1 may be seen. In adults, a study by Silverman has shown that patients with an initial FEV1 lower than 25% of what was predicted benefited the most from magnesium sulfate therapy.
Asthma is classified according to the frequency of symptoms, FEV1 and peak expiratory flow rate.[4]
Classification of asthma severity[4]
Severity
Symptom frequency
Nighttime symptoms
Peak expiratory flow rate or FEV1 of predicted
Variability of peak expiratory flow rate or FEV1
Intermittent
< once a week
≤ twice per month
≥ 80% predicted
< 20%
Mild persistent
> once per week but < once per day
> twice per month
20–30%
Moderate persistent
Daily
> once per week
60–80% predicted
> 30%
Severe persistent
Frequent
< 60% predicted
Bacterial Pneumonia
Some more study resources from Wikipedia
Respiratory sounds: Stridor ·Wheeze ·Rales ·Rhonchi ·Hamman’s sign
Dyspnea ·Hyperventilation/Hypoventilation ·Hyperpnea/Tachypnea/Bradypnea ·Orthopnoea/Platypnea
Biot’s respiration ·Cheyne-Stokes respiration ·Kussmaul breathing
Hiccup ·Mouth breathingAsphyxia ·Cough ·Pleurisy ·Sputum
Respiratory arrest ·Hypercapnia/Hypocapnia
2/18/09 – Dr Hernandez Respiratory Emergencies 1 Control of breathing Involuntary centers start in Pons, Medulla Pons regulates pattern Apneustic center – controls length of inspiration Pneumotaxic center – expiration Central chemoreceptors – separated by blood brain barrier Responds to PaCO2 Diffuses across blood brain barrier Changes PH of CSF Peripheral Chemoreceptors Respond to decreased o2 supply (carotid bodies, aortic arch) Secondary drive – hypoxic drive Low Hb saturation Decreased perfusion Baroreceptors Sense pressure changes respond to low BP hypovolemia hypoxia vs hypoxemia hypoxia – low tissue oxygen hypoxemia – low blood oxygen (sp02 – pulse oximeter) Rate controlled by CO2 level in Medulla Tachypnea vs bradypnea refers to rate Hypopnea vs hyperpnea refers to depth Orthopnea – SOB while lying down Pluerodynia – painful breathing (Platypnea – SOB while sitting up) Dyspnea – subjective description Hyperventilation vs Hypoventilation Hypocarbia – hypercarbia Patient can be tachypneic without hyperventilating Cheyne-Stokes – regularly irregular – alternates between apnea and tachypnea – damage to control centers Biots – Irregular chaotic – severe brain injury or brain stem herniation – abnormal pattern of breathing characterized by groups of quick, shallow inspirations followed by regular or irregular periods of apnea.[ Apneustic Breathing – prolonged inhalation – severe brain injury Kussmaul’s – too much acid, body trying to blow off excess – rapid deep – seen in in DKA, metabolic acidosis Lung Receptors Pulmonary stretch receptors Herring-Breuer Reflex Irritant Receptors Cough receptors Epithelium of tracheal bronchial tree Cause sneeze cough, laryngospasm Vasovagal response (Saddle Embolus – deadly) Normal Spo2 & PaCo2 Spo2 -90-97% acceptable to maintain life, 80s for chronic smokers or COPD – keep neonates, preemies at 93% to prevent o2 damage 35-45mm normal PaCo2 97% of o2 in blood carried by Hb Each Hb takes 4 o2 molecules (a pulse oximeter will read 100% when Hb saturated with 3 o2 molecules) As atmospheric pressure rises you have a higher uptake of 02 – so at high altitudes you will have lower sats You can saturate more o2 in a cold liquid, warm environment requires higher pressure to saturate same o2 70% of co2 transported as bicarbonate, easier to cross membranes, Some Hypercarbia causes – opiates, resp arrest, obstructive diseases, obstructions, etc Any increase in metabolic rate will cause rate to increase and vice versa Initial assessment
2/18/09 – Dr Hernandez
Respiratory Emergencies 1
Control of breathing
Rate controlled by CO2 level in Medulla
Hyperventilation vs Hypoventilation
Patient can be tachypneic without hyperventilating
Lung Receptors
Irritant Receptors
(Saddle Embolus – deadly)
Normal Spo2 & PaCo2
Initial assessment
2/2/09 Functions of the respiratory system Exchange of gases Excretory function (volatile substances – alcohol, garlic, acetone, etc) Acid Base Balance Metabolic and aerobic metabolism Respiratory Physiology Ventilation Hyper/Hypo refers to depth of ventilation and leads to hypo/hyper carbia Eupnea – normal breathing Tachypnea Braydpnea Apnea Dyspnea External Respiration – alveolar capillary membrane (also have bearing on acid/base levels due to transport of co2) Internal Respiration – cellular respiration (also have bearing on acid/base levels due to transport of co2) Transportation of Gases – O2 transported on hemoglobin, (hemoglobin is directly affected by temperature, in cold it picks up O2 but does not release it easily in the tissues – red cheeks because blood is cold O2 stays in the vessels) alkalosis – o2 not given up by Hgb, acidosis- o2 given up by Hgb much faster Regulation – Physics of ventilation Boyle’s Law – describes how air moves in and out of lungs Dalton’s Law of Partial Pressure – distribution of gas Henry’s law of Solubility of gases – describes how gases dissolve in water To be continued….
2/2/09
Functions of the respiratory system
Metabolic and aerobic metabolism
Respiratory Physiology
Physics of ventilation
To be continued….
12/17/08 Nasotracheal Intubation Blind Pt must be breathing spontaneously Indications Conscious patients Possible spinal injury Trismus (clenched teeth) Contraindications Head injury Equipment Smaller size tube No scope Technique Preoxygenate Advise to inhale Lube with KY Aim tip towards ear Position just above glottis opening Auscultate and otherwise confirm placement Digital Intubation No scope Blood Bite block Left hand open epiglottis using index and middle fingers along right side of mouth, advance tube with right hand along left side of mouth Pass tube 2 inches past your fingers Use bite block Not for peds ET Suction Sterile technique May cause arrhythmias and vagal response Avoid unless causing obstruction Pre and post oxygenate Pour sterile water down ET tube Introduce catheter and engage suction on way out for <15 sec Extubation Hyperogygenate Have equipment Confirm responsiveness Lean forward Suction oropharynx Deflate cuff Extubate as pt coughs or exhales Pediatrics Miller blade (floppy epiglottis) Preemie – size 0 Size of pinky or >1 year age/4+4= size Use Braslow tape Smaller size are cuffless Curve stylet upwards, airway more anterior and superior Can cause parasympathetic response – counter with Atropine Sulphate Sniffing position Record depth of tube Confirm proper placement Breath sounds travel easily in children DOPE (troubleshooting a deteriorating patient) Displacement – re-auscultate Obstruction – suction Pneumothorax – absent sounds on one side Equipment failure – CPAP (Continuous Positive Airway Pressure) Used in CHF, obvious difficulty, oxygenated (maintain own airway)- increases pressure in the alveoli to keep fluid in vascular space and out of lungs Contraindications AMS No patent airway COPD More… Connect to o2 tank. Set pressure to 5cm at minimum – increase to 10cm max. Introduce slowly to patient Chest decompression Evacuate air in the pleuritic space Indications (all required) JVD Obvious dyspnea Tracheal deviation Procedure Landmark: 2nd intercostal space – (above third rib – below ribs contains vasculature and nerves) Listen for air escape Create a flutter valve – use a glove finger
12/17/08
Nasotracheal Intubation
Digital Intubation
ET Suction
Extubation
Pediatrics
DOPE (troubleshooting a deteriorating patient)
CPAP (Continuous Positive Airway Pressure)
Chest decompression
Procedure
