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Pediatrics 1
Mar 23rd, 2009 by RH-111
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Pediatric Assessment, Dr Cooper

3/23/09

See www.cpem.org

  • Planning: Triage & transport – Needs vs. resources – enroute, review and plan
  • Arrival: General Impression: Pediatric Assessment Triangle (PAT) – Hands off assessment – ABC Appearance, Work of Breathing, Circulation to skin
  • Initial Assessment: Rapid cardiopulmonary assessment – Hands on
  • Focused History: pertinent negatives, relevant findings

Pediatric Assessment Triangle

  1. Appearance
  • Alertness
  • Distractibility
  • Consolibility
  • Eye contact
  • Quality of cry
  • Spontaneous movement
  • (All critical to whether brain is perfused properly)

     

  1. Work of Breathing
  • Chest rise
  • Rocking motions
  • Retractions
  • Nasal flailing
  • Head bobbing
  • Grunting
  • Snoring
  • Stridor

(C)Circulation

  • Pallor
  • Mottled
  • Blue
  • Grey

 

Initial Assessment: Rapid cardiopulmonary assessment – Hands on

  • Airway –clear? Maintainable?, stable?
  • Breathing – ventilation, oxygenating, stable? In peds rates and effort are not necessarily related like adults, effort much more important!
  • Circulation: Shock? Cardiogenic?, stable? Shock: inability of blood to meet metabolic needs of the tissues- Mental status, pulse rate and character; distal vs. proximal, skin color, BP. Cardiogenic shock: Dysrhythmias, other , compensated, decompensated, cardiopulmonary failure (cardiogenic shock not initially treated with fluid)

Focused History: pertinent negatives, relevant findings

Why peds don’t have heart attacks: no CAD, atherosclerosis, etc –congenital heart diseases are rare. Adults drop dead, kids droop dead (secondary to respiratory arrest, etc)

 

Anatomic & Physiologic differences

Child airway – funnel shaped, narrowest part is at crichoid ring- adult s cylinder, narrowest at glottis

  • Small jaw, large tongue, prone t soft tissue obstruction – reposition
  • Immature immune system. lack of specific antibodies, protective mucus layer
  • Infants are nasal breathers, keep clear
  • Floppy omega shaped epiglottis
  • Narrow subglottic area

Remember if suspected C spine injury, stabilize c spine before/while maintaining airway

Breathing anatomy

  • Adult – diagonal ribs, stiff cartilage, stronger muscles
  • Ped – horizontal ribs, soft cartilage, weaker muscles – diaphragmatic breathers, much less alveoli – faster o2 depletion. Susceptible to barotraumas, high risk of Pneumothorax, bag until chest rise, no more. (head bobbing grunting – near end resp failure)young tissue – high elastin content– shift mediastinum -easily

Breathing assessment requires an open Airway! – ASSESS A, THEN FIX A! THEN GO ON TO B!

  • Is ventilation adequate – inspect chest rise – capability    
  • Auscultation – air entry
  • (Missed slide)

Always consider hypoxia first as cause for AMS

  • Auscultate in armpits, small chest, sounds travel
  • ETT only of BVM ineffective
  • Consider NG/OG if abdominal distention

Circulation

  • Adults, big hearts large chambers and thin walls, Starlings Law (like a spring, recoil helps CO)
  • peds – small chambers thick walls – can’t vary CO well with heart walls, CO depends only on HR
  • Adults – stiff vessels – vigorous response to hypovolemia and hypothermia
  • Peds – soft vessels – more compliant vessels
  • Smaller blood volume, lose lager percentage compared to adult
  • Smaller fat mass – larger relative blood volume

Bleeding control – direct pressure – retain systemic o2

Shock assessment – cause assessment – cardiogenic etc – Simultaneous palpation of central and peripheral pulses – strong central weak peripheral – compensated – everything weak; decompensated shock

  • Tachycardia = 150 – 5x age in year
  • Kids get mottled – not clammy
  • Cap refill – use warm extremities
  • Minimum systolic BP: 80 + 2x age

Adrenaline makes you stupid – use a Broselow Tape

Study Topics
Jan 11th, 2009 by RH-111
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Random topics of interest, not in lectures yet

1/11/09

  • ACE inhibitor (ACEI) blocks conversion of angiotensin I to angiotensin 2 – results in lowering of BP, used to treat HTN & CHF.
    (Mechanism of Action: inhibits angiotensin converting enzyme, interfering w/ conversion of angiotensin I to angiotensin II) RAAS
  • Vasopressin
  • Calcium Channel Blocker (CCB) Most calcium channel blockers decrease the force of contraction of the myocardium (muscle of the heart). This is known as the negative inotropic effect of calcium channel blockers.
  • Pituitary
  • Krebs cycle
  • Diuretic
  • Fremitus
  • Uremia
    is a term used to loosely describe the illness accompanying kidney failure (also called renal failure), in particular the nitrogenous waste products associated with the failure of this organ.

    In kidney failure, urea and other waste products, which are normally excreted into the urine, are retained in the blood. Early symptoms include anorexia and lethargy, and late symptoms can include decreased mental acuity and coma. It is usually diagnosed in kidney dialysis patients when the glomerular filtration rate, a measure of kidney function, is below 50% of normal.[2]

  • Tetany – is a medical sign, the involuntary contraction of muscles, caused by diseases and other conditions that increase the action potential frequency. The muscle cramps caused by the disease tetanus are not classified as tetany; rather, they are due to a blocking of the inhibition to the neurons that supply muscles.
  • Motor_end_plate
  • Cushing’s Triad
  • Pnuemonia
  • Pulmonary embolism (PE)

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